The Foundation supports research across basic, translational and clinical science to speed breakthroughs that can lead to the creation of new treatments and a better quality of life for people with Parkinson's disease.
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Previously funded studies appear chronologically, with the most recent appearing first.
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Rapid Response Innovation Awards, 2009Oligomeric Equilibrium in Male and Female Brains with Alpha-synuclein Pathology
Objective/Rationale:
Men are more susceptible to Parkinson's disease than women. Alpha-synuclein aggregates are a hallmark of the disease and it is increasingly believed that alpha-synuclein... -
Rapid Response Innovation Awards, 2009Characterizing Transgenic Pre-Clinical Models that Express a Mutant LRRK2 Gene Selectively in Adulthood
Objective/Rationale:
Mutations of the gene LRRK2 are identified in both familial and sporadic Parkinson’s disease (PD). How mutation of the LRRK2 gene causes neuronal dysfunction in PD remains to be... -
Postural Instability and Gait Disturbances, 2009Role of the Posterior Parietal Cortex and Pedunculopontine Nucleus in the Pathophysiology of Freezing of Gait: An MRI and TMS Study
Objective/Rationale:
Although disturbances in the posterior parietal cortex (PPC) and pedunculopontine nucleus (PPN) are thought to be involved in the development of freezing of gait (FOG) in PD... -
Cognitive Deficits and Mood Disorders in Parkinson's Disease, 2008A Rodent Model of Pathological Gambling Associated with Medicated Parkinsonian Patients
Objective/Rationale:
The increase in impulsive and risky behaviors, like gambling, hyper-sexuality, and compulsive shopping are emotionally and financially devastating, and impose a significant burden... -
Cognitive Deficits and Mood Disorders in Parkinson's Disease, 2008Cognitive Decline in Parkinson's Disease
Objective/Rationale:
There are many studies on cognitive and behavioral alterations in Parkinson’s disease, but each of these investigations brought only a small piece of a large puzzle. Consequently... -
Program-non-specific Funding, 2008Validation of TorsinA as a Target for PD Therapy in Mammalian Models
Objective/Rationale:
Mutations in the protein torsinA cause a hereditary form of dystonia. While the normal function of torsinA is not entirely clear, it is thought to be a “chaperone,” a molecule...
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Our funding programs support basic, translational and clinical research from academia and industry.