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Funded Studies

The Foundation supports research across basic, translational and clinical science to speed breakthroughs that can lead to the creation of new treatments and a better quality of life for people with Parkinson's disease.

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Previously funded studies appear chronologically, with the most recent appearing first.

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  • Rapid Response Innovation Awards, 2013
    Glycosylation as an Inhibitor of Alpha-synuclein Aggregation

    Objective/Rationale:             
    During the development of Parkinson’s disease the protein alpha-synuclein forms toxic clumps, or aggregates, that have been demonstrated to directly contribute to the...

  • Rapid Response Innovation Awards, 2013
    Investigation of the Contribution of Copy Number Alterations in Parkinson’s Disease Genes to Pathogenesis

    Objective/Rationale:             
    In most cases, the cause of Parkinson’s disease (PD) is unknown. Testing blood for DNA mutations is usually negative, but there is increasing evidence that DNA can...

  • LRRK2 Challenge, 2013
    Functional Analysis of LRRK2 Phosphorylation in Human Dopaminergic Neurodegeneration

    Objective/Rationale:
    Although mutations in LRRK2 are the most common genetic cause of Parkinson’s disease (PD) known, the molecular mechanism is unclear. LRRK2 protein is phosphorylated (addition of a...

  • Research Grant, 2013
    Kinase Activity in Lymphocytes

    Objective/Rationale:       
    It has been suggested that the G2019S mutation in the LRRK2 gene leads to increased kinase (a type of enzyme) activity. Previous work has been conducted in cultured cells...

  • Research Grant, 2013
    Phenylbutyrate for Alpha-synuclein Clearance from the Brain

    Objective/Rationale:             
    In our previous research, we have discovered that a drug called phenylbutyrate can turn on a protective gene in the brain and can prevent Parkinson’s disease (PD) in...

  • Rapid Response Innovation Awards, 2013
    The Role of Phosphorylation and Oligomerixation on Alpha-synuclein-induced Vesicle Rupture

    Objective/Rationale:             
    The propagation of pathological alpha-synuclein in the central nervous system resembles a spreading viral infection. We have observed that alpha-synuclein can enter...

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