2371 - 2380 of 6403 Results
Title
Year
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OPENTitle: Blood DNA methylation profiling identifies cathepsin Z dysregulation in pulmonary arterial hypertensionJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 15Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1038/s41467-023-44683-0Best OA location URL: https://www.nature.com/articles/s41467-023-44683-0.pdfCitation Count: 11
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OPENTitle: APOE genotype dependent molecular abnormalities in the cerebrovasculature of Alzheimer’s disease and age-matched non-demented brainsJournal Name: Molecular BrainPublisher: Springer Science and Business Media LLCVol: 14Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s13041-021-00803-9Best OA location URL: https://molecularbrain.biomedcentral.com/counter/pdf/10.1186/s13041-021-00803-9Citation Count: 24
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OPENTitle: A genetic and transcriptomic assessment of the KTN1 gene in Parkinson’s disease riskJournal Name: Neurobiology of AgingPublisher: Elsevier BVVol: 134Issue #:Start Page: 66End Page: 73Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.neurobiolaging.2023.11.001Best OA location URL: https://pmc.ncbi.nlm.nih.gov/articles/PMC10843739/pdf/nihms-1947313.pdfCitation Count: 0
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OPENTitle: Longitudinal faster anxiety progression of GBA variant carriers in the early Parkinson’s disease cohortJournal Name: Frontiers in NeurosciencePublisher: Frontiers Media SAVol: 18Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3389/fnins.2024.1353759Best OA location URL: https://www.frontiersin.org/articles/10.3389/fnins.2024.1353759/pdf?isPublishedV2=FalseCitation Count: 2
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OPENTitle: A 3′UTR Insertion Is a Candidate Causal Variant at the TMEM106B Locus Associated With Increased Risk for FTLD-TDPJournal Name: Neurology GeneticsPublisher: Ovid Technologies (Wolters Kluwer Health)Vol: 10Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1212/nxg.0000000000200124Best OA location URL: https://www.neurology.org/doi/pdfdirect/10.1212/NXG.0000000000200124Citation Count: 4
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OPENTitle: Improved measurement of disease progression in people living with early Parkinson’s disease using digital health technologiesJournal Name: Communications MedicinePublisher: Springer Science and Business Media LLCVol: 4Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s43856-024-00481-3Best OA location URL: https://www.nature.com/articles/s43856-024-00481-3.pdfCitation Count: 8
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OPENTitle: Downregulation of Dickkopf-3, a Wnt antagonist elevated in Alzheimer’s disease, restores synapse integrity and memory in a disease mouse modelJournal Name: eLifePublisher: eLife Sciences Publications, LtdVol: 12Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.7554/elife.89453Best OA location URL: https://elifesciences.org/articles/89453.bibCitation Count: 8
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OPENTitle: Combinatorial selective ER-phagy remodels the ER during neurogenesisJournal Name: Nature Cell BiologyPublisher: Springer Science and Business Media LLCVol: 26Issue #: 3Start Page: 378End Page: 392Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1038/s41556-024-01356-4Best OA location URL: https://www.nature.com/articles/s41556-024-01356-4.pdfCitation Count: 22
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OPENTitle: Disease progression strikingly differs in research and real-world Parkinson’s populationsJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 10Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1038/s41531-024-00667-5Best OA location URL: https://www.nature.com/articles/s41531-024-00667-5.pdfCitation Count: 8
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OPENTitle: Biallelic NAA60 variants with impaired N-terminal acetylation capacity cause autosomal recessive primary familial brain calcificationsJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 15Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1038/s41467-024-46354-0Best OA location URL: https://escholarship.org/uc/item/8hc1r6m8Citation Count: 21