461 - 470 of 5832 Results
Title
Year
- The Relationship between p-tau217, p-tau231, and p-tau205 in the Human Brain Is Affected by the Cellular Environment and Alzheimer’s Disease Pathology2024OPENTitle: The Relationship between p-tau217, p-tau231, and p-tau205 in the Human Brain Is Affected by the Cellular Environment and Alzheimer’s Disease PathologyJournal Name: CellsPublisher: MDPI AGVol: 13Issue #: 4Start Page: 331End Page: 331Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3390/cells13040331Citation Count: 1
- Efficacy of biologically-directed daylight therapy on sleep and circadian rhythm in Parkinson's disease: a randomised, double-blind, parallel-group, active-controlled, phase 2 clinical trial2024OPENTitle: Efficacy of biologically-directed daylight therapy on sleep and circadian rhythm in Parkinson's disease: a randomised, double-blind, parallel-group, active-controlled, phase 2 clinical trialJournal Name: eClinicalMedicinePublisher: Elsevier BVVol: 69Issue #:Start Page: 102474End Page: 102474Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1016/j.eclinm.2024.102474Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10867415Citation Count: 5
- Microstructural integrity of the locus coeruleus and its tracts reflect noradrenergic degeneration in Alzheimer’s disease and Parkinson’s disease2024OPENTitle: Microstructural integrity of the locus coeruleus and its tracts reflect noradrenergic degeneration in Alzheimer’s disease and Parkinson’s diseaseJournal Name: Translational NeurodegenerationPublisher: Springer Science and Business Media LLCVol: 13Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s40035-024-00400-5Citation Count: 3
- Sequential CRISPR screening reveals partial NatB inhibition as a strategy to mitigate alpha-synuclein levels in human neurons2024OPENTitle: Sequential CRISPR screening reveals partial NatB inhibition as a strategy to mitigate alpha-synuclein levels in human neuronsJournal Name: Science AdvancesPublisher: American Association for the Advancement of Science (AAAS)Vol: 10Issue #: 6Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1126/sciadv.adj4767Citation Count: 4
- A leaky gut dysregulates gene networks in the brain associated with immune activation, oxidative stress, and myelination in a mouse model of colitis2024OPENTitle: A leaky gut dysregulates gene networks in the brain associated with immune activation, oxidative stress, and myelination in a mouse model of colitisJournal Name: Brain, Behavior, and ImmunityPublisher: Elsevier BVVol: 117Issue #:Start Page: 473End Page: 492Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1016/j.bbi.2024.02.007Best OA location URL: https://doi.org/10.1016/j.bbi.2024.02.007Citation Count: 7
- Large-scale proteomics analysis of five brain regions from Parkinson’s disease patients with a GBA1 mutation2024OPENTitle: Large-scale proteomics analysis of five brain regions from Parkinson’s disease patients with a GBA1 mutationJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 10Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-024-00645-xCitation Count: 3
- Associations between Sleep Disorders and Impulsive-Compulsive Behaviors in Parkinson's Disease: A Prospective Cohort Study2024OPENTitle: Associations between Sleep Disorders and Impulsive-Compulsive Behaviors in Parkinson's Disease: A Prospective Cohort StudyJournal Name: NeuroepidemiologyPublisher: S. Karger AGVol: 58Issue #: 4Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1159/000536555Citation Count: 1
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OPENTitle: CURTAIN—A unique web-based tool for exploration and sharing of MS-based proteomics dataJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 121Issue #: 7Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1073/pnas.2312676121Citation Count: 9
- Investigating alpha‐synuclein co‐pathology in Alzheimer's disease by means of cerebrospinal fluid alpha‐synuclein seed amplification assay2024OPENTitle: Investigating alpha‐synuclein co‐pathology in Alzheimer's disease by means of cerebrospinal fluid alpha‐synuclein seed amplification assayJournal Name: Alzheimer's & DementiaPublisher: WileyVol: 20Issue #: 4Start Page: 2444End Page: 2452Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1002/alz.13658Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/alz.13658Citation Count: 24
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OPENTitle: Modulation of α-synuclein in vitro aggregation kinetics by its alternative splice isoformsJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 121Issue #: 7Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1073/pnas.2313465121Best OA location URL: https://www.pnas.org/doi/pdf/10.1073/pnas.2313465121Citation Count: 3