4881 - 4890 of 5832 Results
Title
Year
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OPENTitle: Age-specific penetrance ofLRRK2G2019S in the Michael J. Fox Ashkenazi Jewish LRRK2 ConsortiumJournal Name: NeurologyPublisher: Ovid Technologies (Wolters Kluwer Health)Vol: 85Issue #: 1Start Page: 89End Page: 95Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1212/wnl.0000000000001708Best OA location URL: https://europepmc.org/articles/pmc4501942?pdf=renderCitation Count: 140
- KTKEGV repeat motifs are key mediators of normal α-synuclein tetramerization: Their mutation causes excess monomers and neurotoxicity2015OPENTitle: KTKEGV repeat motifs are key mediators of normal α-synuclein tetramerization: Their mutation causes excess monomers and neurotoxicityJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 112Issue #: 31Start Page: 9596End Page: 9601Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1505953112Best OA location URL: https://www.pnas.org/content/pnas/112/31/9596.full.pdfCitation Count: 140
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OPENTitle: Exercise improves cognition in Parkinson's disease: The PRET-PD randomized, clinical trialJournal Name: Movement DisordersPublisher: WileyVol: 30Issue #: 12Start Page: 1657End Page: 1663Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mds.26291Best OA location URL: https://europepmc.org/articles/pmc4609235?pdf=renderCitation Count: 161
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OPENTitle: When DLB, PD, and PSP masquerade as MSAJournal Name: NeurologyPublisher: Ovid Technologies (Wolters Kluwer Health)Vol: 85Issue #: 5Start Page: 404End Page: 412Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1212/wnl.0000000000001807Best OA location URL: https://n.neurology.org/content/neurology/85/5/404.full.pdfCitation Count: 298
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OPENTitle: Improving the clinical detection of Lewy body dementia with the Lewy body composite risk scoreJournal Name: Alzheimer's & Dementia: Diagnosis, Assessment & Disease MonitoringPublisher: WileyVol: 1Issue #: 3Start Page: 316End Page: 324Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1016/j.dadm.2015.05.004Best OA location URL: http://www.dadm.alzdem.com/article/S2352872915000561/pdfCitation Count: 68
- Neuroimaging Analysis of the Dopamine Basis for Apathetic Behaviors in an MPTP-Lesioned Primate Model2015OPENTitle: Neuroimaging Analysis of the Dopamine Basis for Apathetic Behaviors in an MPTP-Lesioned Primate ModelJournal Name: PLOS ONEPublisher: Public Library of Science (PLoS)Vol: 10Issue #: 7Start Page: e0132064End Page: e0132064Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1371/journal.pone.0132064Best OA location URL: https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0132064&type=printableCitation Count: 23
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OPENTitle: A lipid switch unlocks Parkinson’s disease-associated ATP13A2Journal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 112Issue #: 29Start Page: 9040End Page: 9045Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1508220112Best OA location URL: https://www.pnas.org/content/pnas/112/29/9040.full.pdfCitation Count: 86
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OPENTitle: Neuropathology of Beta-propeller protein associated neurodegeneration (BPAN): a new tauopathyJournal Name: Acta Neuropathologica CommunicationsPublisher: Springer Science and Business Media LLCVol: 3Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s40478-015-0221-3Best OA location URL: https://actaneurocomms.biomedcentral.com/counter/pdf/10.1186/s40478-015-0221-3Citation Count: 49
- Nuclear receptor Nurr1 agonists enhance its dual functions and improve behavioral deficits in an animal model of Parkinson’s disease2015OPENTitle: Nuclear receptor Nurr1 agonists enhance its dual functions and improve behavioral deficits in an animal model of Parkinson’s diseaseJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 112Issue #: 28Start Page: 8756End Page: 8761Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1509742112Best OA location URL: https://www.pnas.org/content/pnas/112/28/8756.full.pdfCitation Count: 165
- Binding to serine 65‐phosphorylated ubiquitin primes Parkin for optimal PINK 1‐dependent phosphorylation and activation2015OPENTitle: Binding to serine 65‐phosphorylated ubiquitin primes Parkin for optimal PINK 1‐dependent phosphorylation and activationJournal Name: EMBO reportsPublisher: Springer Science and Business Media LLCVol: 16Issue #: 8Start Page: 939End Page: 954Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.15252/embr.201540352Best OA location URL: https://doi.org/10.15252/embr.201540352Citation Count: 216