7451 - 7475 of 8404 Results
Title
Year
- Comparison of Parkinson Risk in Ashkenazi Jewish Patients With Gaucher Disease andGBAHeterozygotes2014OPENTitle: Comparison of Parkinson Risk in Ashkenazi Jewish Patients With Gaucher Disease andGBAHeterozygotesJournal Name: JAMA NeurologyPublisher: American Medical Association (AMA)Vol: 71Issue #: 6Start Page: 752End Page: 752Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1001/jamaneurol.2014.313Best OA location URL: https://europepmc.org/articles/pmc4082726?pdf=renderCitation Count: 191
- Control of dopaminergic neuron survival by the unfolded protein response transcription factor XBP12014OPENTitle: Control of dopaminergic neuron survival by the unfolded protein response transcription factor XBP1Journal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 111Issue #: 18Start Page: 6804End Page: 6809Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1321845111Best OA location URL: https://www.pnas.org/content/pnas/111/18/6804.full.pdfCitation Count: 198
- Cross-Sectional and Longitudinal Relationships Between Cerebrospinal Fluid Biomarkers and Cognitive Function in People Without Cognitive Impairment From Across the Adult Life Span2014OPENTitle: Cross-Sectional and Longitudinal Relationships Between Cerebrospinal Fluid Biomarkers and Cognitive Function in People Without Cognitive Impairment From Across the Adult Life SpanJournal Name: JAMA NeurologyPublisher: American Medical Association (AMA)Vol: 71Issue #: 6Start Page: 742End Page: 742Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1001/jamaneurol.2014.445Best OA location URL: https://europepmc.org/articles/pmc4051849?pdf=renderCitation Count: 39
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RESTRICTEDTitle: Enteric GFAP expression and phosphorylation in Parkinson's diseaseJournal Name: Journal of NeurochemistryPublisher: WileyVol: 130Issue #: 6Start Page: 805End Page: 815Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1111/jnc.12742Citation Count: 169
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OPENTitle: MHC-I expression renders catecholaminergic neurons susceptible to T-cell-mediated degenerationJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 5Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/ncomms4633Best OA location URL: https://www.nature.com/articles/ncomms4633.pdfCitation Count: 282
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OPENTitle: Parkinson's disease-linked mutations in VPS35 induce dopaminergic neurodegenerationJournal Name: Human Molecular GeneticsPublisher: Oxford University Press (OUP)Vol: 23Issue #: 17Start Page: 4621End Page: 4638Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1093/hmg/ddu178Best OA location URL: https://academic.oup.com/hmg/article-pdf/23/17/4621/2215861/ddu178.pdfCitation Count: 147
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RESTRICTEDTitle: The neurobiological basis of cognitive impairment in Parkinson's diseaseJournal Name: Movement DisordersPublisher: WileyVol: 29Issue #: 5Start Page: 634End Page: 650Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1002/mds.25857Best OA location URL: https://europepmc.org/articles/pmc4049032?pdf=renderCitation Count: 300
- Positive Reinforcement Mediated by Midbrain Dopamine Neurons Requires D1 and D2 Receptor Activation in the Nucleus Accumbens2014OPENTitle: Positive Reinforcement Mediated by Midbrain Dopamine Neurons Requires D1 and D2 Receptor Activation in the Nucleus AccumbensJournal Name: PLoS ONEPublisher: Public Library of Science (PLoS)Vol: 9Issue #: 4Start Page: e94771End Page: e94771Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1371/journal.pone.0094771Best OA location URL: https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0094771&type=printableCitation Count: 145
- Unaltered Striatal Dopamine Release Levels in Young Parkin Knockout, Pink1 Knockout, DJ-1 Knockout and LRRK2 R1441G Transgenic Mice2014OPENTitle: Unaltered Striatal Dopamine Release Levels in Young Parkin Knockout, Pink1 Knockout, DJ-1 Knockout and LRRK2 R1441G Transgenic MiceJournal Name: PLoS ONEPublisher: Public Library of Science (PLoS)Vol: 9Issue #: 4Start Page: e94826End Page: e94826Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1371/journal.pone.0094826Best OA location URL: https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0094826&type=printableCitation Count: 33
- The Thorny Problem of Dyskinesias: Dendritic Spines, Synaptic Plasticity, and Striatal Dysfunction2014OPENTitle: The Thorny Problem of Dyskinesias: Dendritic Spines, Synaptic Plasticity, and Striatal DysfunctionJournal Name: Biological PsychiatryPublisher: Elsevier BVVol: 75Issue #: 9Start Page: 676End Page: 677Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.biopsych.2014.02.019Best OA location URL: https://europepmc.org/articles/pmc4120525?pdf=renderCitation Count: 1
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RESTRICTEDTitle: Resistance of naturally secreted α‐synuclein to proteolysisJournal Name: The FASEB JournalPublisher: WileyVol: 28Issue #: 7Start Page: 3146End Page: 3158Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1096/fj.13-245852Citation Count: 19
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OPENTitle: Essential Tremor in the Elderly and Risk for DementiaJournal Name: Journal of Neurodegenerative DiseasesPublisher: WileyVol: 2014Issue #:Start Page: 1End Page: 5Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1155/2014/328765Best OA location URL: https://downloads.hindawi.com/archive/2014/328765.pdfCitation Count: 22
- Combined Fenobam and Amantadine Signigficantly Reduce Levodopa-Induced Dyskinesia in the 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP)-Lesioned Primate Model of Parkinson’s Disease (P1.016)2014RESTRICTEDTitle: Combined Fenobam and Amantadine Signigficantly Reduce Levodopa-Induced Dyskinesia in the 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP)-Lesioned Primate Model of Parkinson’s Disease (P1.016)Journal Name:Publisher:Vol:Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier:Citation Count: 0
- Combined Fenobam and Amantadine Signigficantly Reduce Levodopa-Induced Dyskinesia in the 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP)-Lesioned Primate Model of Parkinson’s Disease (P1.016)2014RESTRICTEDTitle: Combined Fenobam and Amantadine Signigficantly Reduce Levodopa-Induced Dyskinesia in the 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP)-Lesioned Primate Model of Parkinson’s Disease (P1.016)Journal Name: NeurologyPublisher: Ovid Technologies (Wolters Kluwer Health)Vol: 82Issue #: 10_supplementStart Page:End Page:Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1212/wnl.82.10_supplement.p1.016Citation Count: 0
- The BioFIND Study (Fox Investigation For New Discovery Of Biomarkers In Parkinson’s Disease): Design And Methodology (P4.043)2014RESTRICTEDTitle: The BioFIND Study (Fox Investigation For New Discovery Of Biomarkers In Parkinson’s Disease): Design And Methodology (P4.043)Journal Name:Publisher:Vol:Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier:Citation Count: 2
- The BioFIND Study (Fox Investigation For New Discovery Of Biomarkers In Parkinson’s Disease): Design And Methodology (P4.043)2014RESTRICTEDTitle: The BioFIND Study (Fox Investigation For New Discovery Of Biomarkers In Parkinson’s Disease): Design And Methodology (P4.043)Journal Name: NeurologyPublisher: Ovid Technologies (Wolters Kluwer Health)Vol: 82Issue #: 10_supplementStart Page:End Page:Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1212/wnl.82.10_supplement.p4.043Citation Count: 5
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OPENTitle: Evaluation of mild cognitive impairment subtypes in Parkinson's diseaseJournal Name: Movement DisordersPublisher: WileyVol: 29Issue #: 6Start Page: 756End Page: 764Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mds.25875Best OA location URL: https://europepmc.org/articles/pmc4013249?pdf=renderCitation Count: 57
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RESTRICTEDTitle: Treating Parkinson's disease in the 21st century: Can stem cell transplantation compete?Journal Name: Journal of Comparative NeurologyPublisher: WileyVol: 522Issue #: 12Start Page: 2802End Page: 2816Publication Date:Open Access(OA) Status: RESTRICTEDLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1002/cne.23577Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/cne.23577Citation Count: 46
- Large-scale resting state network correlates of cognitive impairment in Parkinson's disease and related dopaminergic deficits2014OPENTitle: Large-scale resting state network correlates of cognitive impairment in Parkinson's disease and related dopaminergic deficitsJournal Name: Frontiers in Systems NeurosciencePublisher: Frontiers Media SAVol: 8Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3389/fnsys.2014.00045Best OA location URL: https://www.frontiersin.org/articles/10.3389/fnsys.2014.00045/pdfCitation Count: 100
- Nanomicellar formulation of coenzyme Q10 (Ubisol-Q10) effectively blocks ongoing neurodegeneration in the mouse 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model: potential use as an adjuvant treatment in Parkinson's disease2014OPENTitle: Nanomicellar formulation of coenzyme Q10 (Ubisol-Q10) effectively blocks ongoing neurodegeneration in the mouse 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model: potential use as an adjuvant treatment in Parkinson's diseaseJournal Name: Neurobiology of AgingPublisher: Elsevier BVVol: 35Issue #: 10Start Page: 2329End Page: 2346Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.neurobiolaging.2014.03.032Best OA location URL: https://europepmc.org/articles/pmc4892899?pdf=renderCitation Count: 66
- Accelerating Drug Development for the Field: Building Clinical Trial Recruitment Infrastructure in Parkinson’s2014RESTRICTEDTitle: Accelerating Drug Development for the Field: Building Clinical Trial Recruitment Infrastructure in Parkinson’sJournal Name:Publisher:Vol:Issue #:Start Page: 22End Page: 27Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.14524/cr-13-00066r1.1Citation Count: 1
- Can Isolated Enlarged Virchow-Robin Spaces Influence the Clinical Manifestations of Parkinson's Disease?2014OPENTitle: Can Isolated Enlarged Virchow-Robin Spaces Influence the Clinical Manifestations of Parkinson's Disease?Journal Name: Movement Disorders Clinical PracticePublisher: WileyVol: 1Issue #: 1Start Page: 67End Page: 69Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mdc3.12009Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/mdc3.12009Citation Count: 17
- Leucine-Rich Repeat Kinase 2 Binds to Neuronal Vesicles through Protein Interactions Mediated by Its C-Terminal WD40 Domain2014OPENTitle: Leucine-Rich Repeat Kinase 2 Binds to Neuronal Vesicles through Protein Interactions Mediated by Its C-Terminal WD40 DomainJournal Name: Molecular and Cellular BiologyPublisher: Informa UK LimitedVol: 34Issue #: 12Start Page: 2147End Page: 2161Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1128/mcb.00914-13Best OA location URL: https://mcb.asm.org/content/mcb/34/12/2147.full.pdfCitation Count: 99
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RESTRICTEDTitle: Tau acts as a mediator for Alzheimer's disease‐related synaptic deficitsJournal Name: European Journal of NeurosciencePublisher: WileyVol: 39Issue #: 7Start Page: 1202End Page: 1213Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1111/ejn.12504Best OA location URL: https://europepmc.org/articles/pmc3983570?pdf=renderCitation Count: 44
- Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer‐induced AMPA glutamate receptor signaling deficits2014OPENTitle: Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer‐induced AMPA glutamate receptor signaling deficitsJournal Name: European Journal of NeurosciencePublisher: WileyVol: 39Issue #: 7Start Page: 1214End Page: 1224Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/ejn.12507Best OA location URL: https://europepmc.org/articles/pmc4123852?pdf=renderCitation Count: 119