8151 - 8175 of 8587 Results
Title
Year
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OPENTitle: Team Science Approaches to Unravel Monogenic Parkinson's Disease on a Global ScaleJournal Name: Movement DisordersPublisher: WileyVol: 39Issue #: 10Start Page: 1868End Page: 1873Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1002/mds.29925Best OA location URL: https://movementdisorders.onlinelibrary.wiley.com/doi/pdf/10.1002/mds.29925Citation Count: 3
- Increased burden of rare risk variants across gene expression networks predisposes to sporadic Parkinson’s disease2025OPENTitle: Increased burden of rare risk variants across gene expression networks predisposes to sporadic Parkinson’s diseaseJournal Name: Cell ReportsPublisher: Elsevier BVVol: 44Issue #: 5Start Page: 115636End Page: 115636Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1016/j.celrep.2025.115636Best OA location URL: https://doi.org/10.1016/j.celrep.2025.115636Citation Count: 0
- Aromaticity at position 39 in α‐synuclein: A modulator of amyloid fibril assembly and membrane‐bound conformations2022OPENTitle: Aromaticity at position 39 in α‐synuclein: A modulator of amyloid fibril assembly and membrane‐bound conformationsJournal Name: Protein SciencePublisher: WileyVol: 31Issue #: 7Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-ncDOI - Digital Object Identifier: 10.1002/pro.4360Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/pro.4360Citation Count: 9
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OPENTitle: T lymphocyte senescence is attenuated in Parkinson’s diseaseJournal Name: Journal of NeuroinflammationPublisher: Springer Science and Business Media LLCVol: 18Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1186/s12974-021-02287-9Best OA location URL: https://jneuroinflammation.biomedcentral.com/track/pdf/10.1186/s12974-021-02287-9Citation Count: 31
- Exploring the Experience of Wearing Off in Parkinson’s Disease: A Qualitative Research Approach (P5.8-050)2019RESTRICTEDTitle: Exploring the Experience of Wearing Off in Parkinson’s Disease: A Qualitative Research Approach (P5.8-050)Journal Name: NeurologyPublisher: Ovid Technologies (Wolters Kluwer Health)Vol: 92Issue #: 15_supplementStart Page:End Page:Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1212/wnl.92.15_supplement.p5.8-050Citation Count: 0
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RESTRICTEDTitle: Higher urate in LRRK2 mutation carriers resistant to Parkinson diseaseJournal Name: Annals of NeurologyPublisher: WileyVol: 85Issue #: 4Start Page: 593End Page: 599Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1002/ana.25436Citation Count: 52
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RESTRICTEDTitle: Can We Treat Neurodegenerative Proteinopathies by Enhancing Protein Degradation?Journal Name: Movement DisordersPublisher: WileyVol: 37Issue #: 7Start Page: 1346End Page: 1359Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1002/mds.29058Citation Count: 17
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OPENTitle: 14-3-3 Proteins regulate mutant LRRK2 kinase activity and neurite shorteningJournal Name: Human Molecular GeneticsPublisher: Oxford University Press (OUP)Vol: 25Issue #: 1Start Page: 109End Page: 122Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1093/hmg/ddv453Best OA location URL: https://academic.oup.com/hmg/article-pdf/25/1/109/7145789/ddv453.pdfCitation Count: 78
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OPENTitle: Toward the quantification of α-synuclein aggregates with digital seed amplification assaysJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 121Issue #: 3Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1073/pnas.2312031121Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/10801878Citation Count: 14
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OPENTitle: Pitx3 is required for development of substantia nigra dopaminergic neuronsJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 100Issue #: 7Start Page: 4245End Page: 4250Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.0230529100Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/153078Citation Count: 382
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OPENTitle: Regulation of Physiologic Actions of LRRK2: Focus on AutophagyJournal Name: Neurodegenerative DiseasesPublisher: S. Karger AGVol: 10Issue #: 1-4Start Page: 238End Page: 241Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1159/000332599Best OA location URL: https://www.karger.com/Article/Pdf/332599Citation Count: 39
- Pharmacological Characterization of Membrane-Expressed Human Trace Amine-Associated Receptor 1 (TAAR1) by a Bioluminescence Resonance Energy Transfer cAMP Biosensor2008OPENTitle: Pharmacological Characterization of Membrane-Expressed Human Trace Amine-Associated Receptor 1 (TAAR1) by a Bioluminescence Resonance Energy Transfer cAMP BiosensorJournal Name: Molecular PharmacologyPublisher: Elsevier BVVol: 74Issue #: 3Start Page: 585End Page: 594Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1124/mol.108.048884Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/3766527Citation Count: 147
- Compounds from an unbiased chemical screen reverse both ER-to-Golgi trafficking defects and mitochondrial dysfunction in Parkinson’s disease models2009OPENTitle: Compounds from an unbiased chemical screen reverse both ER-to-Golgi trafficking defects and mitochondrial dysfunction in Parkinson’s disease modelsJournal Name: Disease Models & MechanismsPublisher: The Company of BiologistsVol: 3Issue #: 3-4Start Page: 194End Page: 208Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1242/dmm.004267Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/2869493Citation Count: 178
- The Parkinson’s disease protein alpha-synuclein is a modulator of processing bodies and mRNA stability2022OPENTitle: The Parkinson’s disease protein alpha-synuclein is a modulator of processing bodies and mRNA stabilityJournal Name: CellPublisher: Elsevier BVVol: 185Issue #: 12Start Page: 2035End Page: 2056.e33Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-nd, cc-by-nc-nd, cc-by-nc-ndDOI - Digital Object Identifier: 10.1016/j.cell.2022.05.008Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/9394447Citation Count: 115
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OPENTitle: Open science takes on Parkinson’s diseaseJournal Name: eLifePublisher: eLife Sciences Publications, LtdVol: 10Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.7554/elife.66546Best OA location URL: https://cdn.elifesciences.org/articles/66546/elife-66546-v1.pdfCitation Count: 7
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RESTRICTEDTitle: VAMP2 chaperones α-synuclein in synaptic vesicle co-condensatesJournal Name: Nature Cell BiologyPublisher: Springer Science and Business Media LLCVol: 26Issue #: 8Start Page: 1287End Page: 1295Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1038/s41556-024-01456-1Citation Count: 23
- Burden of disease scenarios for 204 countries and territories, 2022–2050: a forecasting analysis for the Global Burden of Disease Study 20212024OPENTitle: Burden of disease scenarios for 204 countries and territories, 2022–2050: a forecasting analysis for the Global Burden of Disease Study 2021Journal Name: The LancetPublisher: Elsevier BVVol: 403Issue #: 10440Start Page: 2204End Page: 2256Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1016/s0140-6736(24)00685-8Best OA location URL: https://doi.org/10.1016/s0140-6736(24)00685-8Citation Count: 394
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OPENTitle: SAMTOR is anS-adenosylmethionine sensor for the mTORC1 pathwayJournal Name: SciencePublisher: American Association for the Advancement of Science (AAAS)Vol: 358Issue #: 6364Start Page: 813End Page: 818Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1126/science.aao3265Best OA location URL: https://science.sciencemag.org/content/sci/358/6364/813.full.pdfCitation Count: 463
- Dysregulation of microRNA-219 promotes neurodegeneration through post-transcriptional regulation of tau2015OPENTitle: Dysregulation of microRNA-219 promotes neurodegeneration through post-transcriptional regulation of tauJournal Name: Journal of Clinical InvestigationPublisher: American Society for Clinical InvestigationVol: 125Issue #: 2Start Page: 681End Page: 686Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1172/jci78421Citation Count: 194
- Rapid iPSC inclusionopathy models shed light on formation, consequence, and molecular subtype of α-synuclein inclusions2024OPENTitle: Rapid iPSC inclusionopathy models shed light on formation, consequence, and molecular subtype of α-synuclein inclusionsJournal Name: NeuronPublisher: Elsevier BVVol: 112Issue #: 17Start Page: 2886End Page: 2909.e16Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1016/j.neuron.2024.06.002Best OA location URL: https://doi.org/10.1016/j.neuron.2024.06.002Citation Count: 11
- JNK2 and JNK3 combined are essential for apoptosis in dopamine neurons of the substantia nigra, but are not required for axon degeneration2008OPENTitle: JNK2 and JNK3 combined are essential for apoptosis in dopamine neurons of the substantia nigra, but are not required for axon degenerationJournal Name: Journal of NeurochemistryPublisher: WileyVol: 107Issue #: 6Start Page: 1578End Page: 1588Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/j.1471-4159.2008.05713.xBest OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1111/j.1471-4159.2008.05713.x?download=trueCitation Count: 79
- Hsp104 antagonizes α-synuclein aggregation and reduces dopaminergic degeneration in a rat model of Parkinson disease2008OPENTitle: Hsp104 antagonizes α-synuclein aggregation and reduces dopaminergic degeneration in a rat model of Parkinson diseaseJournal Name: Journal of Clinical InvestigationPublisher: American Society for Clinical InvestigationVol: 118Issue #: 9Start Page: 3087End Page: 3097Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1172/jci35781Best OA location URL: http://www.jci.org/articles/view/35781/files/pdfCitation Count: 195
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OPENTitle: EndoMAP.v1 charts the structural landscape of human early endosome complexesJournal Name: NaturePublisher: Springer Science and Business Media LLCVol: 643Issue #: 8070Start Page: 252End Page: 261Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1038/s41586-025-09059-yBest OA location URL: https://www.nature.com/articles/s41586-025-09059-y.pdfCitation Count: 0
- Evaluation of the PREDIGT score’s performance in identifying newly diagnosed Parkinson’s patients without motor examination2022OPENTitle: Evaluation of the PREDIGT score’s performance in identifying newly diagnosed Parkinson’s patients without motor examinationJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 8Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-022-00360-5Best OA location URL: https://www.nature.com/articles/s41531-022-00360-5.pdfCitation Count: 4
- Role for NUP62 depletion and PYK2 redistribution in dendritic retraction resulting from chronic stress2014OPENTitle: Role for NUP62 depletion and PYK2 redistribution in dendritic retraction resulting from chronic stressJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 111Issue #: 45Start Page: 16130End Page: 16135Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1418896111Best OA location URL: https://www.pnas.org/content/pnas/111/45/16130.full.pdfCitation Count: 50