The Foundation supports research across basic, translational and clinical science to speed breakthroughs that can lead to the creation of new treatments and a better quality of life for people with Parkinson's disease.
Search or browse funded studies
Previously funded studies appear chronologically, with the most recent appearing first.
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ASAP CRN 2025 RFA Scientific Track, 2026Decoding Alpha-Synuclein Conformational Diversity to Enable Advanced Predictive Amplification Assays
Study Rationale:
Our Team’s research shows that special forms of α-synuclein protein extracted or amplified from biospecimen donations from people with Parkinson’s disease...
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ASAP CRN 2025 RFA Scientific Track, 2026Defining the Neural Mechanisms Underlying Heterogeneity in Sleep and Circadian Rhythm Disturbances in Parkinson’s Disease
Study Rationale:
People affected by Parkinson’s experience a range of sleep problems such as excessive daytime sleeping and difficulty sleeping at night. These issues can make...
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ASAP CRN 2025 RFA Scientific Track, 2026Seeded Aggregation of Alpha-Synuclein
Study Rationale:
Filamentous aggregates of the protein alpha-synuclein underlie several neurodegenerative diseases, called synucleinopathies. Parkinson’s disease (PD) is the most common...
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ASAP CRN 2025 RFA Scientific Track, 2026Aging and the Heterogeneity of Parkison’s Disease – The Role of Heterochromatin Erosion
Study Rationale:
Age is the single most significant risk factor for developing Parkinson’s disease (PD), but the mechanisms by which aging contributes to PD are not well...
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ASAP CRN 2025 RFA Scientific Track, 2026CLEAR-PD: Clearance via Lymphatics & Endocytic Alpha-Synuclein Receptors in Parkinson’s Disease
Study Rationale:
In Parkinson’s disease (PD), it is clear that the alpha-synuclein protein accumulates in specific neurons, thereby damaging them to cause neurological symptoms...
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ASAP CRN 2025 RFA Scientific Track, 2026Decoding Co-Pathological Mechanisms in Parkinson's Disease: From Molecular Interactions to Disease Progression
Study Rationale:
Parkinson's disease involves multiple abnormal proteins—α-synuclein, tau, and TDP-43—that clump together in brain cells. These protein clumps don't act alone; they work...
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Our funding programs support basic, translational and clinical research from academia and industry.