91 - 100 of 8100 Results
Title
Year
- Changes in Action Tremor in Parkinson's Disease over Time: Clinical and Neuroimaging Correlates2024OPENTitle: Changes in Action Tremor in Parkinson's Disease over Time: Clinical and Neuroimaging CorrelatesJournal Name: Movement DisordersPublisher: WileyVol: 40Issue #: 2Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1002/mds.30081Citation Count: 0
- Mitochondrial oxidant stress promotes α-synuclein aggregation and spreading in mice with mutated glucocerebrosidase2024OPENTitle: Mitochondrial oxidant stress promotes α-synuclein aggregation and spreading in mice with mutated glucocerebrosidaseJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 10Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1038/s41531-024-00842-8Citation Count: 0
- –Structural Changes in Early-stage Parkinson’s Disease with Resting Tremor at Node, Edge and Network Level2024OPENTitle: –Structural Changes in Early-stage Parkinson’s Disease with Resting Tremor at Node, Edge and Network LevelJournal Name: Brain Research BulletinPublisher: Elsevier BVVol: 220Issue #:Start Page: 111169End Page: 111169Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1016/j.brainresbull.2024.111169Citation Count: 0
-
OPENTitle: A simulated annealing algorithm for randomizing weighted networksJournal Name: Nature Computational SciencePublisher: Springer Science and Business Media LLCVol: 5Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s43588-024-00735-zCitation Count: 0
- The amplification of alpha‐synuclein amyloid fibrils is suppressed under fully quiescent conditions2024OPENTitle: The amplification of alpha‐synuclein amyloid fibrils is suppressed under fully quiescent conditionsJournal Name: Angewandte Chemie International EditionPublisher: WileyVol: 64Issue #: 7Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-ncDOI - Digital Object Identifier: 10.1002/anie.202419173Citation Count: 0
-
OPENTitle: Bidirectional relationship between olfaction and Parkinson’s diseaseJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 10Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-024-00838-4Citation Count: 0
-
OPENTitle: Endo-IP and lyso-IP toolkit for endolysosomal profiling of human-induced neuronsJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 121Issue #: 52Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1073/pnas.2419079121Citation Count: 0
- Temporal control of acute protein aggregate turnover by UBE3C and NRF1-dependent proteasomal pathways2024OPENTitle: Temporal control of acute protein aggregate turnover by UBE3C and NRF1-dependent proteasomal pathwaysJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 121Issue #: 50Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1073/pnas.2417390121Citation Count: 0
- The ZFHX3 GGC Repeat Expansion Underlying Spinocerebellar Ataxia Type 4 has a Common Ancestral Founder2024OPENTitle: The ZFHX3 GGC Repeat Expansion Underlying Spinocerebellar Ataxia Type 4 has a Common Ancestral FounderJournal Name: Movement DisordersPublisher: WileyVol: 40Issue #: 2Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1002/mds.30077Citation Count: 0
- Arrayed CRISPR libraries for the genome-wide activation, deletion and silencing of human protein-coding genes2024OPENTitle: Arrayed CRISPR libraries for the genome-wide activation, deletion and silencing of human protein-coding genesJournal Name: Nature Biomedical EngineeringPublisher: Springer Science and Business Media LLCVol: 9Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41551-024-01278-4Citation Count: 1