5981 - 5990 of 8571 Results
Title
Year
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OPENTitle: CaMKII regulates diacylglycerol lipase-α and striatal endocannabinoid signalingJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 16Issue #: 4Start Page: 456End Page: 463Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nn.3353Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/3636998Citation Count: 74
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OPENTitle: Genetic variability in the regulation of gene expression in ten regions of the human brainJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 17Issue #: 10Start Page: 1418End Page: 1428Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nn.3801Best OA location URL: http://doi.org/10.1038/nn.3801Citation Count: 656
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OPENTitle: Mind matters: placebo enhances reward learning in Parkinson's diseaseJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 17Issue #: 12Start Page: 1793End Page: 1797Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nn.3842Best OA location URL: http://doi.org/10.1038/nn.3842Citation Count: 78
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OPENTitle: Subthalamic nucleus stimulation reverses mediofrontal influence over decision thresholdJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 14Issue #: 11Start Page: 1462End Page: 1467Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nn.2925Citation Count: 652
- Enhancers active in dopamine neurons are a primary link between genetic variation and neuropsychiatric disease2018OPENTitle: Enhancers active in dopamine neurons are a primary link between genetic variation and neuropsychiatric diseaseJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 21Issue #: 10Start Page: 1482End Page: 1492Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/s41593-018-0223-0Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/6334654Citation Count: 95
- Preparation of biogenic gas vesicle nanostructures for use as contrast agents for ultrasound and MRI2017OPENTitle: Preparation of biogenic gas vesicle nanostructures for use as contrast agents for ultrasound and MRIJournal Name: Nature ProtocolsPublisher: Springer Science and Business Media LLCVol: 12Issue #: 10Start Page: 2050End Page: 2080Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nprot.2017.081Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/6185898Citation Count: 143
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OPENTitle: Characterization of a selective inhibitor of the Parkinson's disease kinase LRRK2Journal Name: Nature Chemical BiologyPublisher: Springer Science and Business Media LLCVol: 7Issue #: 4Start Page: 203End Page: 205Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nchembio.538Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/3287420Citation Count: 416
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OPENTitle: Parkinson's disease induced pluripotent stem cells with triplication of the α-synuclein locusJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 2Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-saDOI - Digital Object Identifier: 10.1038/ncomms1453Best OA location URL: https://www.nature.com/articles/ncomms1453.pdfCitation Count: 445
- Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation2015OPENTitle: Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiationJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 6Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/ncomms8314Best OA location URL: https://www.nature.com/articles/ncomms8314.pdfCitation Count: 280
- O-GlcNAc modification blocks the aggregation and toxicity of the protein α-synuclein associated with Parkinson's disease2015OPENTitle: O-GlcNAc modification blocks the aggregation and toxicity of the protein α-synuclein associated with Parkinson's diseaseJournal Name: Nature ChemistryPublisher: Springer Science and Business Media LLCVol: 7Issue #: 11Start Page: 913End Page: 920Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nchem.2361Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/4618406Citation Count: 253