8651 - 8660 of 9131 Results
Title
Year
-
OPENTitle: Is Gauchian genotyping of GBA1 variants reliable?Journal Name: Communications BiologyPublisher: Springer Science and Business Media LLCVol: 8Issue #: 1Start Page: 718End Page: 718Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s42003-025-08059-yBest OA location URL: https://www.nature.com/articles/s42003-025-08059-y.pdfCitation Count: 5
-
OPENTitle: Urinary proteome profiling for stratifying patients with familial Parkinson’s diseaseJournal Name: EMBO Molecular MedicinePublisher: Springer Science and Business Media LLCVol: 13Issue #: 3Start Page: e13257End Page: e13257Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.15252/emmm.202013257Best OA location URL: https://doi.org/10.15252/emmm.202013257Citation Count: 167
-
OPENTitle: The experience of care partners of patients with Parkinson’s disease psychosisJournal Name: PLOS ONEPublisher: Public Library of Science (PLoS)Vol: 16Issue #: 3Start Page: e0248968End Page: e0248968Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1371/journal.pone.0248968Best OA location URL: https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0248968&type=printableCitation Count: 10
- Defining the causes of sporadic Parkinson’s disease in the global Parkinson’s genetics program (GP2)2023OPENTitle: Defining the causes of sporadic Parkinson’s disease in the global Parkinson’s genetics program (GP2)Journal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 9Issue #: 1Start Page: 131End Page: 131Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-023-00533-wBest OA location URL: https://www.nature.com/articles/s41531-023-00533-w.pdfCitation Count: 39
-
OPENTitle: Genetic meta-analysis of levodopa induced dyskinesia in Parkinson’s diseaseJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 9Issue #: 1Start Page: 128End Page: 128Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-023-00573-2Best OA location URL: https://www.nature.com/articles/s41531-023-00573-2.pdfCitation Count: 12
-
OPENTitle: Temporal control of acute protein aggregate turnover by UBE3C and NRF1-dependent proteasomal pathwaysJournal Name: Proceedings of the National Academy of SciencesPublisher: National Academy of SciencesVol: 121Issue #: 50Start Page: e2417390121End Page: e2417390121Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1073/pnas.2417390121Best OA location URL: https://www.pnas.org/doi/pdf/10.1073/pnas.2417390121Citation Count: 7
-
OPENTitle: Autophagic stress activates distinct compensatory secretory pathways in neuronsJournal Name: Proceedings of the National Academy of SciencesPublisher: National Academy of SciencesVol: 122Issue #: 28Start Page: e2421886122End Page: e2421886122Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1073/pnas.2421886122Best OA location URL: https://www.pnas.org/doi/pdf/10.1073/pnas.2421886122Citation Count: 20
-
OPENTitle: Bidirectional relationship between olfaction and Parkinson’s diseaseJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 10Issue #: 1Start Page: 232End Page: 232Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-024-00838-4Best OA location URL: https://doi.org/10.1038/s41531-024-00838-4Citation Count: 4
-
OPENTitle: Biochemical Fractionation of Human α-Synuclein in a Drosophila Model of SynucleinopathiesJournal Name: International Journal of Molecular SciencesPublisher: MDPI AGVol: 25Issue #: 7Start Page: 3643End Page: 3643Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3390/ijms25073643Best OA location URL: https://www.mdpi.com/1422-0067/25/7/3643/pdf?version=1711354407Citation Count: 0
-
OPENTitle: Genetic modulation of mitochondrial NAD+ regeneration does not prevent dopaminergic neuron dysfunction caused by mitochondrial complex I impairmentJournal Name: Frontiers in Cell and Developmental BiologyPublisher: Frontiers Media SAVol: 13Issue #:Start Page: 1650462End Page: 1650462Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3389/fcell.2025.1650462Best OA location URL: https://www.frontiersin.org/journals/cell-and-developmental-biology/articles/10.3389/fcell.2025.1650462/pdfCitation Count: 1